Ca2+ Channels

As yet, the mechanisms of basophil activation induced by IL-3 and GM-CSF have not been fully explored, but the demonstration that this histamine-releasing activities of these two cytokines are less than additive suggests that they share the same biochemical pathways

As yet, the mechanisms of basophil activation induced by IL-3 and GM-CSF have not been fully explored, but the demonstration that this histamine-releasing activities of…

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Ca2+ Channels

Specific diagnoses, such as for example indication for biologic therapy, had not been available because of limited recording of International Classification of Illnesses (ICD) rules in digital health data

Specific diagnoses, such as for example indication for biologic therapy, had not been available because of limited recording of International Classification of Illnesses (ICD) rules…

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Ca2+ Channels

The pRITE_MT_CID_V5_puro_3myc_CD4 plasmid employed for RITE was constructed by combining several synthesized gBlock DNA fragments (IDT) and a PCR-fragment containing the CD4 gene in to the NotI/PmeI site of the pMT-CID-V5 plasmid (for sequence points, see Supplementary Table?1)

The pRITE_MT_CID_V5_puro_3myc_CD4 plasmid employed for RITE was constructed by combining several synthesized gBlock DNA fragments (IDT) and a PCR-fragment containing the CD4 gene in to…

Comments Off on The pRITE_MT_CID_V5_puro_3myc_CD4 plasmid employed for RITE was constructed by combining several synthesized gBlock DNA fragments (IDT) and a PCR-fragment containing the CD4 gene in to the NotI/PmeI site of the pMT-CID-V5 plasmid (for sequence points, see Supplementary Table?1)
Ca2+ Channels

Previous work discovered that lack of Gin4 and Hsl1 causes cells to endure unrestrained growth throughout a extended mitotic arrest, which implies a role is played by them in linking cell cycle progression to cell growth

Previous work discovered that lack of Gin4 and Hsl1 causes cells to endure unrestrained growth throughout a extended mitotic arrest, which implies a role is…

Comments Off on Previous work discovered that lack of Gin4 and Hsl1 causes cells to endure unrestrained growth throughout a extended mitotic arrest, which implies a role is played by them in linking cell cycle progression to cell growth